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Atrial fibrillation as an autoimmune disease?

  • Michael H. Gollob
    Correspondence
    Address reprint requests and correspondence: Dr Michael H. Gollob, Division of Cardiology and the Arrhythmia Research Laboratory, University of Ottawa Heart Institute, 40 Ruskin St, Room 3228, Ottawa, Ontario, Canada K1Y 4W7
    Affiliations
    Division of Cardiology and the Arrhythmia Research Laboratory, University of Ottawa Heart Institute, Ottawa, Ontario, Canada
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Published:January 21, 2013DOI:https://doi.org/10.1016/j.hrthm.2013.01.023
      The paradoxical behavior of the human body to both self-repair and self-destruct is indeed a fascinating enigma. The ability of bones to mend, lacerations to seal, and the usual resistance to severe microbial infection is a direct result of biochemistry and physiology aimed to protect and promote our health and longevity. In contrast, this same physiology appears to have the potential to attack one’s own body and lead to the development of chronic disease.
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